Lyme disease is not caused by a microorganism

Reposted from Amandha Vollmers Telegram Channel: https://t.me/amandhavollmer

The system has been poisoned. Usually by vaccines, chronic antibiotic use, other heavy metals, microplastics, traumas or nutritional deficiencies, as we see with literally all disease states. 

Giving more antibiotics may prove temporary relief as the microorganisms are active in trying to remove the waste from the body and the symptoms are produced in an attempt to release the wastes. 

Improper treatment occurs due to false diagnosis as nutritional stores are not replenished and microbial support is not given.

An individual who is on the edge of a purge only needs one event to trigger them into an attempted release. That could be a shock, a bite from any organism, another poisoning from a pharmaceutical, even improper eating. We see this with states of fibromyalgia and chronic fatigue, as well. These individuals are ticking time bombs and it only takes one trigger to set them off where the body now needs to go into a cleansing cycle which produces symptoms that people judge as negative. 

There is your Lyme disease, there is your covid, there is your long covid, there is your <insert any disease you want that is blamed on microorganisms and viruses.>

Borrelia studies

Isolation of Borrelia burgdorferi sensu lato from blood of adult patients with borrelial lymphocytoma, Lyme neuroborreliosis, Lyme arthritis and acrodermatitis chronica atrophicans

https://sci-hub.st/10.1007/s15010-010-0062-8

Results: “Borrelia burgdorferi sensu lato was isolated from the blood of 1/53 (1.9%) patients with borrelial lymphocytoma, 6/176 (3.4%) patients with Lyme neuroborreliosis, 1/13 (7.7%) patients with Lyme arthritis, and 3/ 200 (1.5%) patients with acrodermatitis chronica atrophicans.”

“At the time of the blood culture, erythema migrans was present in 4/11 (36.4%) borrelia blood culture-positive patients …”

Isolation of Borrelia Spirochetes from Patients in Texas

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC269164/

Results: “During this study, blood specimens from 100 patients were cultured for B. burgdorferi. We cultivated spirochetes from two (2%) specimens: one chilled whole blood and one frozen clot. A total of 31 skin specimens were placed in culture, resulting in 8 (26%) isolates, and spirochetes were observed in 1 of 7 (14%) cultured CSF specimens. Tissues from two autopsies (kidney, liver, spleen, lung, and lymph node) were submitted, and spirochetes were detected in the liver and spleen of one patient. Spirochetes were isolated from one of three (33%) joint fluids (knee) and from the one bone specimen that was submitted (wrist).”

Here’s a summary of the paper on the alleged causative agent of lyme disease: 

– Here is the first paper from 1982 describing experiments with the ‘isolated’ bacterium Borrelia burgdorferi: https://escholarship.org/content/qt9vj3t37b/qt9vj3t37b.pdf?t=p4g42c

– Only epidemiological evidence is referred to as causality, there is no direct evidence that or how the bacterium triggers the symptoms. 

– The clinical symptoms are described as diffuse: an inflammatory disease that is initially noticeable through skin leasions. It was first recognized as a form of inflammatory arthritis in Lyme, Connecticut, although the clinical disease was also observed in other states. 

– In order to prove the causality, experiments were then carried out with 8 rabbits. 30-40 ticks were taped to the shaved abdomen of each rabbit. That would be comparable to a person weighing 185lb (83 kg) having over 1,000 ticks stuck to their stomach. (after weight comparison). 

– The bacterium was NEVER detected directly in the rabbits, only by means of antibody levels, which are not specific, as the authors themselves show, since the antibody tests always react, just in different dilutions. 

– The antibody test used also reacts positively in people who do not suffer from the disease, only in lower concentrations.

 – It is also important that these experiments were carried out without scientific controls. (e.g. rabbit ticks without bacteria, same procedure)

– A pure isolate of the bacterium was not obtained or used, so it is not possible to distinguish whether the clinical symptoms (skin inflammation) were caused by the bite wounds of the ticks or by the bacterium.

Willy Burgdorfer: Lyme disease (1996)  

https://sci-hub.st/10.1016/s0736-4679(96)00143-6

”Laboratory diagnosis of Lyme disease remains problematic. Although Borrelia burgdorferi organisms have been identified in biopsy specimens of erythema migrans lesions (IS), such biopsy is not practical for emergency physicians and many other clinicians. Serologic testing is of very limited value in acute cases, inasmuch as IgM levels do not begin to rise for 2-4 weeks following the onset of infection (17). Immunological tests such as enzyme-linked immunosorbent assay (ELBA) and immunofluorescence assay suffer from poor reproducibility of positive and negative results (IO). Western immunoblotting, however, may be useful in identifying false-positive ELISA results (19). Consequently, currently there is no standard laboratory test diagnostic for Lyme disease (12), and the diagnosis is best made on clinical and epidemiologic grounds.”

[…]

”Chronic Lyme disease may be overdiagnosed, often on the basis of false-positive serology …”

Recent Progress in Lyme Disease and Remaining Challenges

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8416313/

“The diagnosis of LD [lyme disease] can be a complex task for the provider because, outside of the erythema migrans (EM) lesion of early LD, diagnosis relies on non-specific clinical signs and symptoms that may or may not be supported by laboratory evidence.”

[…]

“LD is known as the “great imitator” because symptoms are varied and often overlap with common health complaints, sometimes making early diagnosis more difficult. … There are likely hundreds of health conditions with significant overlap with these non-specific signs and symptoms.”

Hollstrom E: Penicillin treatment of erythema chronicum migrans Afzelius

https://www.medicaljournals.se/acta/content_files/files/pdf/38/5/38285289.pdf

Thank you to a colleague for amassing the research.